The genitourinary tract is one of the most common sites of extrapulmonary tuberculosis, accounting for 15-20% of infections outside lungs and approximately 4-8% with pulmonary tuberculosis will develop significant genitourinary tuberculosis [1]. The diagnosis is made on basis of urine culture studies or histopathologic analysis and is supported by radiologic studies. The findings of the disease on plain abdominal radiography are calcifications. Renal calcifications are a one of the common manifestations of tuberculosis at conventional radiography, occurring in 24-44% [2]. Intravenous urography may show a variety of findings, including moth-eaten calyces, amputated infundibula, hydronephrosis or hydronephroureter due to ureteral strictures and non-function of a kidney [3, 4]. CT is the most sensitive modality for renal calcifications which occur in over 50% of cases of genitourinary tuberculosis [2, 5] and is thought to be a mainstay in the diagnostic images of renal tuberculosis, showing renal parenchymal cavity, mass and scaring, local parenchymal thinning, stricture of infundibula [4, 6]. CT urography is a relatively new imaging examination which can provide comprehensive evaluation of both renal parenchyma and urothelium. Thus, sings of ureteral and bladder involvement by the disease may be nicely depicted [3]. The most useful radiologic feature of urinary tract tuberculosis is the multiplicity of abnormal findings [2], therefore the diagnosis of the disease can be suggested by a single examination (CT urography). Although MRI is reportedly of limited value in diagnostic images, it has an ability to depict the characteristic features corresponding to the subtle pathological changes and has some advantages including no radiation burden, detective capability of dilated collecting system, calyx and ureteral strictures independent of renal function [3, 7]. In present case, these findings of local parenchymal thinning and dilated calices might be pathologically ascribed to characteristic tubercular changes, not only inflammation, fibrosis and destructive caseous necrosis in the renal parenchyma, but the stricture by fibrosis at ureter and/or calyx infundibula. Furthermore, the nodule in the renal parenchyma had isointensity on T1-weighted images and low intensity on T2-weighted images without gadolinium enhancement. This nodule seemed to correspond to a caseous necrotic granuloma found in the resected specimen. Thus MRI provided us a clue to further examinations in focusing on urinary tract tuberculosis leading to successful diagnosis. Although CT is indeed the most common modality in diagnostic images, the diagnosis cannot be made by a single examination and requires multi-modality methods [4]. Present case suggests that MRI could provide more informative features of renal tuberculosis, especially in case of no calcifications.